METTL14 is required for exercise-induced cardiac hypertrophy and protects against myocardial ischemia-reperfusion injury
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DOI: 10.1038/s41467-022-34434-y
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- Ralf Gilsbach & Martin Schwaderer & Sebastian Preissl & Björn A. Grüning & David Kranzhöfer & Pedro Schneider & Thomas G. Nührenberg & Sonia Mulero-Navarro & Dieter Weichenhan & Christian Braun & Mart, 2018. "Distinct epigenetic programs regulate cardiac myocyte development and disease in the human heart in vivo," Nature Communications, Nature, vol. 9(1), pages 1-14, December.
- Xiang Wang & Jing Feng & Yuan Xue & Zeyuan Guan & Delin Zhang & Zhu Liu & Zhou Gong & Qiang Wang & Jinbo Huang & Chun Tang & Tingting Zou & Ping Yin, 2016. "Structural basis of N6-adenosine methylation by the METTL3–METTL14 complex," Nature, Nature, vol. 534(7608), pages 575-578, June.
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- Tianbao Ye & Cheng Chen & Di Wang & Chengjie Huang & Zhiwen Yan & Yu Chen & Xian Jin & Xiuyuan Wang & Xianting Ding & Chengxing Shen, 2024. "Protective effects of Pt-N-C single-atom nanozymes against myocardial ischemia-reperfusion injury," Nature Communications, Nature, vol. 15(1), pages 1-19, December.
- Na Chen & Lizhe Guo & Lu Wang & Sisi Dai & Xiaocheng Zhu & E. Wang, 2024. "Sleep fragmentation exacerbates myocardial ischemia‒reperfusion injury by promoting copper overload in cardiomyocytes," Nature Communications, Nature, vol. 15(1), pages 1-17, December.
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