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Biological Models of Carcinogenesis and Quantitative Cancer Risk Assessment

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  • Suresh H. Moolgavkar

Abstract

Biologically‐based models of carcinogenesis were originally developed to explain certain quanti‐tative phenomena associated with carcinogenesis, and to provide a framework within which questions regarding the process could be addressed. Some limitations in the use of these models for quantitative cancer risk assessment are discussed.

Suggested Citation

  • Suresh H. Moolgavkar, 1994. "Biological Models of Carcinogenesis and Quantitative Cancer Risk Assessment," Risk Analysis, John Wiley & Sons, vol. 14(6), pages 879-882, December.
    • Handle: RePEc:wly:riskan:v:14:y:1994:i:6:p:879-882
    DOI: 10.1111/j.1539-6924.1994.tb00049.x
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    References listed on IDEAS

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    1. E. Georg Luebeck & Suresh H. Moolgavkar, 1991. "Stochastic Analysis of Intermediate Lesions in Carcinogenesis Experiments," Risk Analysis, John Wiley & Sons, vol. 11(1), pages 149-157, March.
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    Cited by:

    1. Ravi P. Subramaniam & Chao Chen & Kenny S. Crump & Danielle DeVoney & John F. Fox & Christopher J. Portier & Paul M. Schlosser & Chad M. Thompson & Paul White, 2008. "Uncertainties in Biologically‐Based Modeling of Formaldehyde‐Induced Respiratory Cancer Risk: Identification of Key Issues," Risk Analysis, John Wiley & Sons, vol. 28(4), pages 907-923, August.

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