Pharmacological inhibition of HDAC6 improves muscle phenotypes in dystrophin-deficient mice by downregulating TGF-β via Smad3 acetylation
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DOI: 10.1038/s41467-022-34831-3
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References listed on IDEAS
- Masatoshi Nomura & En Li, 1998. "Smad2 role in mesoderm formation, left–right patterning and craniofacial development," Nature, Nature, vol. 393(6687), pages 786-790, June.
- Rik Derynck & Ying E. Zhang, 2003. "Smad-dependent and Smad-independent pathways in TGF-β family signalling," Nature, Nature, vol. 425(6958), pages 577-584, October.
- Wenting Guo & Maximilian Naujock & Laura Fumagalli & Tijs Vandoorne & Pieter Baatsen & Ruben Boon & Laura Ordovás & Abdulsamie Patel & Marc Welters & Thomas Vanwelden & Natasja Geens & Tine Tricot & V, 2017. "HDAC6 inhibition reverses axonal transport defects in motor neurons derived from FUS-ALS patients," Nature Communications, Nature, vol. 8(1), pages 1-15, December.
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- Caroline Dour & Maria Chatzifrangkeskou & Coline Macquart & Maria M. Magiera & Cécile Peccate & Charlène Jouve & Laura Virtanen & Tiina Heliö & Katriina Aalto-Setälä & Silvia Crasto & Bruno Cadot & Dé, 2022. "Actin-microtubule cytoskeletal interplay mediated by MRTF-A/SRF signaling promotes dilated cardiomyopathy caused by LMNA mutations," Nature Communications, Nature, vol. 13(1), pages 1-21, December.
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