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Feedback regulation of steady-state epithelial turnover and organ size

Author

Listed:
  • Jackson Liang

    (Stanford University School of Medicine)

  • Shruthi Balachandra

    (Stanford University School of Medicine)

  • Sang Ngo

    (Stanford University School of Medicine)

  • Lucy Erin O’Brien

    (Stanford University School of Medicine)

Abstract

Steady-state turnover of the Drosophila midgut arises through an intercellular, E-cadherin–EGFR relay that couples the death of individual enterocytes to the divisions of nearby stem cells.

Suggested Citation

  • Jackson Liang & Shruthi Balachandra & Sang Ngo & Lucy Erin O’Brien, 2017. "Feedback regulation of steady-state epithelial turnover and organ size," Nature, Nature, vol. 548(7669), pages 588-591, August.
    • Handle: RePEc:nat:nature:v:548:y:2017:i:7669:d:10.1038_nature23678
    DOI: 10.1038/nature23678
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    Cited by:

    1. Peng Zhang & Stephen M. Pronovost & Marco Marchetti & Chenge Zhang & Xiaoyu Kang & Tahmineh Kandelouei & Christopher Li & Bruce A. Edgar, 2024. "Inter-cell type interactions that control JNK signaling in the Drosophila intestine," Nature Communications, Nature, vol. 15(1), pages 1-14, December.
    2. Xiaotong Li & Jason Karpac, 2023. "A distinct Acyl-CoA binding protein (ACBP6) shapes tissue plasticity during nutrient adaptation in Drosophila," Nature Communications, Nature, vol. 14(1), pages 1-17, December.

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