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Global translational reprogramming is a fundamental layer of immune regulation in plants

Author

Listed:
  • Guoyong Xu

    (Howard Hughes Medical Institute-Gordon and Betty Moore Foundation, Duke University)

  • George H. Greene

    (Howard Hughes Medical Institute-Gordon and Betty Moore Foundation, Duke University)

  • Heejin Yoo

    (Howard Hughes Medical Institute-Gordon and Betty Moore Foundation, Duke University)

  • Lijing Liu

    (Howard Hughes Medical Institute-Gordon and Betty Moore Foundation, Duke University)

  • Jorge Marqués

    (Howard Hughes Medical Institute-Gordon and Betty Moore Foundation, Duke University)

  • Jonathan Motley

    (Howard Hughes Medical Institute-Gordon and Betty Moore Foundation, Duke University)

  • Xinnian Dong

    (Howard Hughes Medical Institute-Gordon and Betty Moore Foundation, Duke University)

Abstract

Global translatome analysis shows that plants also modify their translational output—independently of the changes in transcriptional output—to establish pattern-triggered immunity.

Suggested Citation

  • Guoyong Xu & George H. Greene & Heejin Yoo & Lijing Liu & Jorge Marqués & Jonathan Motley & Xinnian Dong, 2017. "Global translational reprogramming is a fundamental layer of immune regulation in plants," Nature, Nature, vol. 545(7655), pages 487-490, May.
    • Handle: RePEc:nat:nature:v:545:y:2017:i:7655:d:10.1038_nature22371
    DOI: 10.1038/nature22371
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    Cited by:

    1. Zhen Wang & Xiaofan Zhang & Chunyan Liu & Susan Duncan & Runlai Hang & Jing Sun & Lilan Luo & Yiliang Ding & Xiaofeng Cao, 2024. "AtPRMT3-RPS2B promotes ribosome biogenesis and coordinates growth and cold adaptation trade-off," Nature Communications, Nature, vol. 15(1), pages 1-14, December.
    2. Wenya Ma & Yanan Tian & Leping Shi & Jing Liang & Qimeng Ouyang & Jianglong Li & Hongyang Chen & Hongyue Sun & Haoyu Ji & Xu Liu & Wei Huang & Xinlu Gao & Xiaoyan Jin & Xiuxiu Wang & Yining Liu & Yang, 2024. "N-Acetyltransferase 10 represses Uqcr11 and Uqcrb independently of ac4C modification to promote heart regeneration," Nature Communications, Nature, vol. 15(1), pages 1-19, December.

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