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Correction: Corrigendum: Fatty acid carbon is essential for dNTP synthesis in endothelial cells

Author

Listed:
  • Sandra Schoors
  • Ulrike Bruning
  • Rindert Missiaen
  • Karla C. S. Queiroz
  • Gitte Borgers
  • Ilaria Elia
  • Annalisa Zecchin
  • Anna Rita Cantelmo
  • Stefan Christen
  • Jermaine Goveia
  • Ward Heggermont
  • Lucica Goddë
  • Stefan Vinckier
  • Paul P. Van Veldhoven
  • Guy Eelen
  • Luc Schoonjans
  • Holger Gerhardt
  • Mieke Dewerchin
  • Myriam Baes
  • Katrien De Bock
  • Bart Ghesquière
  • Sophia Y. Lunt
  • Sarah-Maria Fendt
  • Peter Carmeliet

Abstract

Nature 520, 192–197 (2015); doi:10.1038/nature14362 We thank our colleagues from the metabolism community (Emile Van Schaftingen and Guido Bommer, University of Louvain, Belgium, and Frans Schuit, University of Leuven, Belgium), who alerted us to a possible confusion arising from our Article. In particular, the statement in the abstract that “Isotope labelling studies in control endothelial cells showed that fatty acid carbons substantially replenished the Krebs cycle” and similar phrases later in the text could be misunderstood as implying anaplerosis.

Suggested Citation

  • Sandra Schoors & Ulrike Bruning & Rindert Missiaen & Karla C. S. Queiroz & Gitte Borgers & Ilaria Elia & Annalisa Zecchin & Anna Rita Cantelmo & Stefan Christen & Jermaine Goveia & Ward Heggermont & L, 2015. "Correction: Corrigendum: Fatty acid carbon is essential for dNTP synthesis in endothelial cells," Nature, Nature, vol. 526(7571), pages 144-144, October.
  • Handle: RePEc:nat:nature:v:526:y:2015:i:7571:d:10.1038_nature14624
    DOI: 10.1038/nature14624
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    1. Nieves Montenegro-Navarro & Claudia García-Báez & Melissa García-Caballero, 2023. "Molecular and metabolic orchestration of the lymphatic vasculature in physiology and pathology," Nature Communications, Nature, vol. 14(1), pages 1-18, December.
    2. Kuei-Pin Chung & Chih-Ning Cheng & Yi-Jung Chen & Chia-Lang Hsu & Yen-Lin Huang & Min-Shu Hsieh & Han-Chun Kuo & Ya-Ting Lin & Yi-Hsiu Juan & Kiichi Nakahira & Yen-Fu Chen & Wei-Lun Liu & Sheng-Yuan R, 2024. "Alveolar epithelial cells mitigate neutrophilic inflammation in lung injury through regulating mitochondrial fatty acid oxidation," Nature Communications, Nature, vol. 15(1), pages 1-23, December.
    3. Balkrishna Chaube & Kathryn M. Citrin & Mahnaz Sahraei & Abhishek K. Singh & Diego Saenz Urturi & Wen Ding & Richard W. Pierce & Raaisa Raaisa & Rebecca Cardone & Richard Kibbey & Carlos Fernández-Her, 2023. "Suppression of angiopoietin-like 4 reprograms endothelial cell metabolism and inhibits angiogenesis," Nature Communications, Nature, vol. 14(1), pages 1-21, December.
    4. Vincent Geldhof & Laura P. M. H. Rooij & Liliana Sokol & Jacob Amersfoort & Maxim Schepper & Katerina Rohlenova & Griet Hoste & Adriaan Vanderstichele & Anne-Marie Delsupehe & Edoardo Isnaldi & Naima , 2022. "Single cell atlas identifies lipid-processing and immunomodulatory endothelial cells in healthy and malignant breast," Nature Communications, Nature, vol. 13(1), pages 1-19, December.
    5. Odeta Meçe & Diede Houbaert & Maria-Livia Sassano & Tania Durré & Hannelore Maes & Marco Schaaf & Sanket More & Maarten Ganne & Melissa García-Caballero & Mila Borri & Jelle Verhoeven & Madhur Agrawal, 2022. "Lipid droplet degradation by autophagy connects mitochondria metabolism to Prox1-driven expression of lymphatic genes and lymphangiogenesis," Nature Communications, Nature, vol. 13(1), pages 1-18, December.
    6. Anastasiya Strembitska & Gwenaël Labouèbe & Alexandre Picard & Xavier P. Berney & David Tarussio & Maxime Jan & Bernard Thorens, 2022. "Lipid biosynthesis enzyme Agpat5 in AgRP-neurons is required for insulin-induced hypoglycemia sensing and glucagon secretion," Nature Communications, Nature, vol. 13(1), pages 1-15, December.
    7. Ling Tao & Mahmoud A. Mohammad & Giorgio Milazzo & Myrthala Moreno-Smith & Tajhal D. Patel & Barry Zorman & Andrew Badachhape & Blanca E. Hernandez & Amber B. Wolf & Zihua Zeng & Jennifer H. Foster & , 2022. "MYCN-driven fatty acid uptake is a metabolic vulnerability in neuroblastoma," Nature Communications, Nature, vol. 13(1), pages 1-17, December.

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