Podocyte OTUD5 alleviates diabetic kidney disease through deubiquitinating TAK1 and reducing podocyte inflammation and injury
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DOI: 10.1038/s41467-024-49854-1
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- Jun Ninomiya-Tsuji & Kazuya Kishimoto & Atsushi Hiyama & Jun-ichiro Inoue & Zhaodan Cao & Kunihiro Matsumoto, 1999. "The kinase TAK1 can activate the NIK-IκB as well as the MAP kinase cascade in the IL-1 signalling pathway," Nature, Nature, vol. 398(6724), pages 252-256, March.
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- Li-Kai Chu & Xu Cao & Lin Wan & Qiang Diao & Yu Zhu & Yu Kan & Li-Li Ye & Yi-Ming Mao & Xing-Qiang Dong & Qian-Wei Xiong & Ming-Cui Fu & Ting Zhang & Hui-Ting Zhou & Shi-Zhong Cai & Zhou-Rui Ma & Ssu-, 2023. "Autophagy of OTUD5 destabilizes GPX4 to confer ferroptosis-dependent kidney injury," Nature Communications, Nature, vol. 14(1), pages 1-17, December.
- Yifei Zhong & Kyung Lee & Yueyi Deng & Yueming Ma & Yiping Chen & Xueling Li & Chengguo Wei & Shumin Yang & Tianming Wang & Nicholas J. Wong & Alecia N. Muwonge & Evren U. Azeloglu & Weijia Zhang & Bh, 2019. "Arctigenin attenuates diabetic kidney disease through the activation of PP2A in podocytes," Nature Communications, Nature, vol. 10(1), pages 1-15, December.
- Hui Sun & Hui Li & Jie Yan & Xiangdong Wang & Mengyuan Xu & Mingxia Wang & Baozhen Fan & Jieying Liu & Ninghua Lin & Xin Wang & Li Li & Shengtian Zhao & Yongfeng Gong, 2022. "Loss of CLDN5 in podocytes deregulates WIF1 to activate WNT signaling and contributes to kidney disease," Nature Communications, Nature, vol. 13(1), pages 1-18, December.
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