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Oscillations of Delta-like1 regulate the balance between differentiation and maintenance of muscle stem cells

Author

Listed:
  • Yao Zhang

    (Developmental Biology/Signal Transduction, Max-Delbrück-Center for Molecular Medicine)

  • Ines Lahmann

    (Developmental Biology/Signal Transduction, Max-Delbrück-Center for Molecular Medicine)

  • Katharina Baum

    (Mathematical Modelling of Cellular Processes, Max-Delbrück-Center for Molecular Medicine
    Hasso Plattner Institute, Digital Engineering Faculty, University of Potsdam)

  • Hiromi Shimojo

    (Institute for Frontier Life and Medical Sciences, Kyoto University
    Osaka University)

  • Philippos Mourikis

    (Univ Paris Est Creteil, INSERM, IMRB)

  • Jana Wolf

    (Mathematical Modelling of Cellular Processes, Max-Delbrück-Center for Molecular Medicine
    Department of Mathematics and Computer Science, Free University Berlin)

  • Ryoichiro Kageyama

    (Institute for Frontier Life and Medical Sciences, Kyoto University)

  • Carmen Birchmeier

    (Developmental Biology/Signal Transduction, Max-Delbrück-Center for Molecular Medicine
    Neurowissenschaftliches Forschungzentrum, NeuroCure Cluster of Excellence, Charité-Universitätsmedizin Berlin)

Abstract

Cell-cell interactions mediated by Notch are critical for the maintenance of skeletal muscle stem cells. However, dynamics, cellular source and identity of functional Notch ligands during expansion of the stem cell pool in muscle growth and regeneration remain poorly characterized. Here we demonstrate that oscillating Delta-like 1 (Dll1) produced by myogenic cells is an indispensable Notch ligand for self-renewal of muscle stem cells in mice. Dll1 expression is controlled by the Notch target Hes1 and the muscle regulatory factor MyoD. Consistent with our mathematical model, our experimental analyses show that Hes1 acts as the oscillatory pacemaker, whereas MyoD regulates robust Dll1 expression. Interfering with Dll1 oscillations without changing its overall expression level impairs self-renewal, resulting in premature differentiation of muscle stem cells during muscle growth and regeneration. We conclude that the oscillatory Dll1 input into Notch signaling ensures the equilibrium between self-renewal and differentiation in myogenic cell communities.

Suggested Citation

  • Yao Zhang & Ines Lahmann & Katharina Baum & Hiromi Shimojo & Philippos Mourikis & Jana Wolf & Ryoichiro Kageyama & Carmen Birchmeier, 2021. "Oscillations of Delta-like1 regulate the balance between differentiation and maintenance of muscle stem cells," Nature Communications, Nature, vol. 12(1), pages 1-16, December.
  • Handle: RePEc:nat:natcom:v:12:y:2021:i:1:d:10.1038_s41467-021-21631-4
    DOI: 10.1038/s41467-021-21631-4
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    Cited by:

    1. Zhang, Yuan & Cao, Jinde & Liu, Lixia & Liu, Haihong & Li, Zhouhong, 2024. "Complex role of time delay in dynamical coordination of neural progenitor fate decisions mediated by Notch pathway," Chaos, Solitons & Fractals, Elsevier, vol. 180(C).

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