Author
Listed:
- Katherine H. Schreiber
(Buck Institute for Research on Aging)
- Sebastian I. Arriola Apelo
(University of Wisconsin-Madison
University of Wisconsin-Madison
William S. Middleton Memorial Veterans Hospital)
- Deyang Yu
(University of Wisconsin-Madison
William S. Middleton Memorial Veterans Hospital
University of Wisconsin-Madison)
- Jacqueline A. Brinkman
(University of Wisconsin-Madison
William S. Middleton Memorial Veterans Hospital)
- Michael C. Velarde
(Aeonian Pharmaceuticals, Inc.
University of the Philippines)
- Faizan A. Syed
(University of Wisconsin-Madison
William S. Middleton Memorial Veterans Hospital)
- Chen-Yu Liao
(Buck Institute for Research on Aging)
- Emma L. Baar
(University of Wisconsin-Madison
William S. Middleton Memorial Veterans Hospital)
- Kathryn A. Carbajal
(University of Wisconsin-Madison
William S. Middleton Memorial Veterans Hospital)
- Dawn S. Sherman
(University of Wisconsin-Madison
William S. Middleton Memorial Veterans Hospital)
- Denise Ortiz
(Buck Institute for Research on Aging
Dominican University)
- Regina Brunauer
(Buck Institute for Research on Aging
Texas A&M University)
- Shany E. Yang
(University of Wisconsin-Madison
William S. Middleton Memorial Veterans Hospital)
- Stelios T. Tzannis
(Aeonian Pharmaceuticals, Inc.)
- Brian K. Kennedy
(Buck Institute for Research on Aging)
- Dudley W. Lamming
(University of Wisconsin-Madison
William S. Middleton Memorial Veterans Hospital
University of Wisconsin-Madison
University of Wisconsin Carbone Cancer Center)
Abstract
Rapamycin, an inhibitor of mechanistic Target Of Rapamycin Complex 1 (mTORC1), extends lifespan and shows strong potential for the treatment of age-related diseases. However, rapamycin exerts metabolic and immunological side effects mediated by off-target inhibition of a second mTOR-containing complex, mTOR complex 2. Here, we report the identification of DL001, a FKBP12-dependent rapamycin analog 40x more selective for mTORC1 than rapamycin. DL001 inhibits mTORC1 in cell culture lines and in vivo in C57BL/6J mice, in which DL001 inhibits mTORC1 signaling without impairing glucose homeostasis and with substantially reduced or no side effects on lipid metabolism and the immune system. In cells, DL001 efficiently represses elevated mTORC1 activity and restores normal gene expression to cells lacking a functional tuberous sclerosis complex. Our results demonstrate that highly selective pharmacological inhibition of mTORC1 can be achieved in vivo, and that selective inhibition of mTORC1 significantly reduces the side effects associated with conventional rapalogs.
Suggested Citation
Katherine H. Schreiber & Sebastian I. Arriola Apelo & Deyang Yu & Jacqueline A. Brinkman & Michael C. Velarde & Faizan A. Syed & Chen-Yu Liao & Emma L. Baar & Kathryn A. Carbajal & Dawn S. Sherman & D, 2019.
"A novel rapamycin analog is highly selective for mTORC1 in vivo,"
Nature Communications, Nature, vol. 10(1), pages 1-12, December.
Handle:
RePEc:nat:natcom:v:10:y:2019:i:1:d:10.1038_s41467-019-11174-0
DOI: 10.1038/s41467-019-11174-0
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Citations
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Cited by:
- Gautham Yepuri & Lisa M. Ramirez & Gregory G. Theophall & Sergei V. Reverdatto & Nosirudeen Quadri & Syed Nurul Hasan & Lei Bu & Devi Thiagarajan & Robin Wilson & Raquel López Díez & Paul F. Gugger & , 2023.
"DIAPH1-MFN2 interaction regulates mitochondria-SR/ER contact and modulates ischemic/hypoxic stress,"
Nature Communications, Nature, vol. 14(1), pages 1-25, December.
- Marine Berquez & Zhiyong Chen & Beatrice Paola Festa & Patrick Krohn & Svenja Aline Keller & Silvia Parolo & Mikhail Korzinkin & Anna Gaponova & Endre Laczko & Enrico Domenici & Olivier Devuyst & Ales, 2023.
"Lysosomal cystine export regulates mTORC1 signaling to guide kidney epithelial cell fate specialization,"
Nature Communications, Nature, vol. 14(1), pages 1-21, December.
- Zhenzhen Zi & Zhuzhen Zhang & Qiang Feng & Chiho Kim & Xu-Dong Wang & Philipp E. Scherer & Jinming Gao & Beth Levine & Yonghao Yu, 2022.
"Quantitative phosphoproteomic analyses identify STK11IP as a lysosome-specific substrate of mTORC1 that regulates lysosomal acidification,"
Nature Communications, Nature, vol. 13(1), pages 1-12, December.
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