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Dipeptidyl Peptidase-4, Heart Failure and Obesity- Current Status and Perspective

Author

Listed:
  • Carmine Finelli

    (Department of Internal Medicine, Italy)

  • Simone Dal Sasso

    (Independent Researcher, Italy)

Abstract

Given that obesity increases the amount of blood that must be supplied to peripheral tissue, it is linked to a higher chronic cardiac workload. The increased demands of increased lean body mass are primarily responsible for the high cardiac output, which is sustained by an increase in ventricular mass, a high normal heart rate, and an increased stroke volume [1]. An increase in non-muscular tissue, which contributes to the development of electrical abnormalities, heart failure, and sudden death, is also implied by an increase in left ventricular (LV) mass [1]. One of the main risk factors for heart failure is obesity itself.

Suggested Citation

  • Carmine Finelli & Simone Dal Sasso, 2024. "Dipeptidyl Peptidase-4, Heart Failure and Obesity- Current Status and Perspective," Biomedical Journal of Scientific & Technical Research, Biomedical Research Network+, LLC, vol. 54(5), pages 46439-46440, February.
    • Handle: RePEc:abf:journl:v:54:y:2024:i:5:p:46439-46440
    DOI: 10.26717/BJSTR.2024.54.008616
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