SLC25A39 is necessary for mitochondrial glutathione import in mammalian cells
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DOI: 10.1038/s41586-021-04025-w
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Cited by:
- Nathan P. Ward & Sang Jun Yoon & Tyce Flynn & Amanda M. Sherwood & Maddison A. Olley & Juliana Madej & Gina M. DeNicola, 2024. "Mitochondrial respiratory function is preserved under cysteine starvation via glutathione catabolism in NSCLC," Nature Communications, Nature, vol. 15(1), pages 1-17, December.
- Xiaojian Shi & Bryn Reinstadler & Hardik Shah & Tsz-Leung To & Katie Byrne & Luanna Summer & Sarah E. Calvo & Olga Goldberger & John G. Doench & Vamsi K. Mootha & Hongying Shen, 2022. "Combinatorial GxGxE CRISPR screen identifies SLC25A39 in mitochondrial glutathione transport linking iron homeostasis to OXPHOS," Nature Communications, Nature, vol. 13(1), pages 1-15, December.
- Izabella A. Pena & Jeffrey S. Shi & Sarah M. Chang & Jason Yang & Samuel Block & Charles H. Adelmann & Heather R. Keys & Preston Ge & Shveta Bathla & Isabella H. Witham & Grzegorz Sienski & Angus C. N, 2025. "SLC25A38 is required for mitochondrial pyridoxal 5’-phosphate (PLP) accumulation," Nature Communications, Nature, vol. 16(1), pages 1-19, December.
- Isshin Shiiba & Naoki Ito & Hijiri Oshio & Yuto Ishikawa & Takahiro Nagao & Hiroki Shimura & Kyu-Wan Oh & Eiki Takasaki & Fuya Yamaguchi & Ryoan Konagaya & Hisae Kadowaki & Hideki Nishitoh & Takehito , 2025. "ER-mitochondria contacts mediate lipid radical transfer via RMDN3/PTPIP51 phosphorylation to reduce mitochondrial oxidative stress," Nature Communications, Nature, vol. 16(1), pages 1-18, December.
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