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ERF mutations reveal a balance of ETS factors controlling prostate oncogenesis

Author

Listed:
  • Rohit Bose

    (Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center
    Memorial Sloan Kettering Cancer Center)

  • Wouter R. Karthaus

    (Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center)

  • Joshua Armenia

    (Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center
    Center for Molecular Oncology, Memorial Sloan Kettering Cancer Center)

  • Wassim Abida

    (Memorial Sloan Kettering Cancer Center)

  • Phillip J. Iaquinta

    (Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center)

  • Zeda Zhang

    (Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center
    Louis V. Gerstner, Jr. Graduate School of Biomedical Sciences, Memorial Sloan Kettering Cancer Center)

  • John Wongvipat

    (Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center)

  • Elizabeth V. Wasmuth

    (Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center)

  • Neel Shah

    (Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center
    Louis V. Gerstner, Jr. Graduate School of Biomedical Sciences, Memorial Sloan Kettering Cancer Center)

  • Patrick S. Sullivan

    (Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center)

  • Michael G. Doran

    (Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center)

  • Ping Wang

    (Albert Einstein College of Medicine)

  • Anna Patruno

    (Memorial Sloan Kettering Cancer Center)

  • Yilin Zhao

    (Albert Einstein College of Medicine)

  • Deyou Zheng

    (Albert Einstein College of Medicine
    Albert Einstein College of Medicine
    Albert Einstein College of Medicine)

  • Nikolaus Schultz

    (Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center
    Center for Molecular Oncology, Memorial Sloan Kettering Cancer Center)

  • Charles L. Sawyers

    (Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center
    Howard Hughes Medical Institute)

Abstract

In prostate cancer, the oncogenicity of transcription factor ERG is mediated, in part, by competition with another member of the ETS family, ERF.

Suggested Citation

  • Rohit Bose & Wouter R. Karthaus & Joshua Armenia & Wassim Abida & Phillip J. Iaquinta & Zeda Zhang & John Wongvipat & Elizabeth V. Wasmuth & Neel Shah & Patrick S. Sullivan & Michael G. Doran & Ping W, 2017. "ERF mutations reveal a balance of ETS factors controlling prostate oncogenesis," Nature, Nature, vol. 546(7660), pages 671-675, June.
    • Handle: RePEc:nat:nature:v:546:y:2017:i:7660:d:10.1038_nature22820
    DOI: 10.1038/nature22820
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    Cited by:

    1. Sheng-You Liao & Dmytro Rudoy & Sander B. Frank & Luan T. Phan & Olga Klezovitch & Julian Kwan & Ilsa Coleman & Michael C. Haffner & Dapei Li & Peter S. Nelson & Andrew Emili & Valeri Vasioukhin, 2023. "SND1 binds to ERG and promotes tumor growth in genetic mouse models of prostate cancer," Nature Communications, Nature, vol. 14(1), pages 1-16, December.

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