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Identification of an atypical monocyte and committed progenitor involved in fibrosis

Author

Listed:
  • Takashi Satoh

    (Laboratory of Host Defense, World Premier Institute Immunology Frontier Research Center, Osaka University
    Research Institute for Microbial Diseases (RIMD), Osaka University)

  • Katsuhiro Nakagawa

    (Laboratory of Host Defense, World Premier Institute Immunology Frontier Research Center, Osaka University
    Research Institute for Microbial Diseases (RIMD), Osaka University)

  • Fuminori Sugihara

    (Laboratory of Biofunctional Imaging, World Premier Institute Immunology Frontier Research Center, Osaka University)

  • Ryusuke Kuwahara

    (Research Center for Ultra-high Voltage Electron Microscopy, Osaka University)

  • Motooki Ashihara

    (Chugai Pharmaceutical Co.)

  • Fumihiro Yamane

    (Laboratory of Host Defense, World Premier Institute Immunology Frontier Research Center, Osaka University
    Research Institute for Microbial Diseases (RIMD), Osaka University)

  • Yosuke Minowa

    (Chugai Pharmaceutical Co.)

  • Kiyoharu Fukushima

    (Laboratory of Host Defense, World Premier Institute Immunology Frontier Research Center, Osaka University
    Research Institute for Microbial Diseases (RIMD), Osaka University)

  • Isao Ebina

    (Laboratory of Host Defense, World Premier Institute Immunology Frontier Research Center, Osaka University
    Research Institute for Microbial Diseases (RIMD), Osaka University
    Chugai Pharmaceutical Co.)

  • Yoshichika Yoshioka

    (Laboratory of Biofunctional Imaging, World Premier Institute Immunology Frontier Research Center, Osaka University)

  • Atsushi Kumanogoh

    (Core Research for Evolutional Science and Technology, Japan Agency for Medical Research and Development
    Allergy and Rheumatic Diseases, Graduate School of Medicine, Osaka University)

  • Shizuo Akira

    (Laboratory of Host Defense, World Premier Institute Immunology Frontier Research Center, Osaka University
    Research Institute for Microbial Diseases (RIMD), Osaka University)

Abstract

An atypical monocyte with partial granulocyte characteristics is identified and shown to be critical for the development of fibrosis.

Suggested Citation

  • Takashi Satoh & Katsuhiro Nakagawa & Fuminori Sugihara & Ryusuke Kuwahara & Motooki Ashihara & Fumihiro Yamane & Yosuke Minowa & Kiyoharu Fukushima & Isao Ebina & Yoshichika Yoshioka & Atsushi Kumanog, 2017. "Identification of an atypical monocyte and committed progenitor involved in fibrosis," Nature, Nature, vol. 541(7635), pages 96-101, January.
    • Handle: RePEc:nat:nature:v:541:y:2017:i:7635:d:10.1038_nature20611
    DOI: 10.1038/nature20611
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    Cited by:

    1. Seiji Taniguchi & Takahiro Matsui & Kenji Kimura & Soichiro Funaki & Yu Miyamoto & Yutaka Uchida & Takao Sudo & Junichi Kikuta & Tetsuya Hara & Daisuke Motooka & Yu-Chen Liu & Daisuke Okuzaki & Eiichi, 2023. "In vivo induction of activin A-producing alveolar macrophages supports the progression of lung cell carcinoma," Nature Communications, Nature, vol. 14(1), pages 1-14, December.

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