IDEAS home Printed from https://ideas.repec.org/a/nat/nature/v515y2014i7526d10.1038_nature13973.html
   My bibliography  Save this article

Radial glia require PDGFD–PDGFRβ signalling in human but not mouse neocortex

Author

Listed:
  • Jan H. Lui

    (University of California, San Francisco, San Francisco, California 94143, USA
    Present addresses: Department of Biology and Howard Hughes Medical Institute, Stanford University, Stanford, California 94305, USA (J.H.L.); Gladstone Institute of Neurological Disease, San Francisco, California 94158, USA (A.J.).)

  • Tomasz J. Nowakowski

    (University of California, San Francisco, San Francisco, California 94143, USA)

  • Alex A. Pollen

    (University of California, San Francisco, San Francisco, California 94143, USA)

  • Ashkan Javaherian

    (University of California, San Francisco, San Francisco, California 94143, USA
    Present addresses: Department of Biology and Howard Hughes Medical Institute, Stanford University, Stanford, California 94305, USA (J.H.L.); Gladstone Institute of Neurological Disease, San Francisco, California 94158, USA (A.J.).)

  • Arnold R. Kriegstein

    (University of California, San Francisco, San Francisco, California 94143, USA)

  • Michael C. Oldham

    (University of California, San Francisco, San Francisco, California 94143, USA)

Abstract

The transcriptional profiles of radial glia are compared between humans and mice during neurogenesis, implicating the growth factor PDGFD and its receptor, PDGFRβ, in human but not mouse neocortical development.

Suggested Citation

  • Jan H. Lui & Tomasz J. Nowakowski & Alex A. Pollen & Ashkan Javaherian & Arnold R. Kriegstein & Michael C. Oldham, 2014. "Radial glia require PDGFD–PDGFRβ signalling in human but not mouse neocortex," Nature, Nature, vol. 515(7526), pages 264-268, November.
    • Handle: RePEc:nat:nature:v:515:y:2014:i:7526:d:10.1038_nature13973
    DOI: 10.1038/nature13973
    as

    Download full text from publisher

    File URL: https://www.nature.com/articles/nature13973
    File Function: Abstract
    Download Restriction: Access to the full text of the articles in this series is restricted.
    File URL: https://libkey.io/10.1038/nature13973?utm_source=ideas
    LibKey link: if access is restricted and if your library uses this service, LibKey will redirect you to where you can use your library subscription to access this item
    ---><---

    As the access to this document is restricted, you may want to search for a different version of it.

    More about this item

    Statistics

    Access and download statistics

    Corrections

    All material on this site has been provided by the respective publishers and authors. You can help correct errors and omissions. When requesting a correction, please mention this item's handle: RePEc:nat:nature:v:515:y:2014:i:7526:d:10.1038_nature13973. See general information about how to correct material in RePEc.

    If you have authored this item and are not yet registered with RePEc, we encourage you to do it here. This allows to link your profile to this item. It also allows you to accept potential citations to this item that we are uncertain about.

    We have no bibliographic references for this item. You can help adding them by using this form .

    If you know of missing items citing this one, you can help us creating those links by adding the relevant references in the same way as above, for each refering item. If you are a registered author of this item, you may also want to check the "citations" tab in your RePEc Author Service profile, as there may be some citations waiting for confirmation.

    For technical questions regarding this item, or to correct its authors, title, abstract, bibliographic or download information, contact: Sonal Shukla or Springer Nature Abstracting and Indexing (email available below). General contact details of provider: http://www.nature.com .

    Please note that corrections may take a couple of weeks to filter through the various RePEc services.

    IDEAS is a RePEc service. RePEc uses bibliographic data supplied by the respective publishers.