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Type I interferon responses in rhesus macaques prevent SIV infection and slow disease progression

Author

Listed:
  • Netanya G. Sandler

    (Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health
    Present address: Division of Infectious Diseases, Department of Internal Medicine, University of Texas Medical Branch at Galveston, Galveston, Texas 77555, USA.)

  • Steven E. Bosinger

    (Emory Vaccine Center, Yerkes National Primate Research Center
    Non-Human Primate Genomics Core, Yerkes National Primate Research Center, Robert W. Woodruff Health Sciences Center, Emory University)

  • Jacob D. Estes

    (AIDS and Cancer Virus Program, Leidos Biomedical Research, Inc., Frederick National Laboratory, Frederick, Maryland 21702, USA)

  • Richard T. R. Zhu

    (Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health)

  • Gregory K. Tharp

    (Emory Vaccine Center, Yerkes National Primate Research Center
    Non-Human Primate Genomics Core, Yerkes National Primate Research Center, Robert W. Woodruff Health Sciences Center, Emory University)

  • Eli Boritz

    (Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health)

  • Doron Levin

    (Weizmann Institute of Science, Rehovot 76100, Israel)

  • Sathi Wijeyesinghe

    (Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health)

  • Krystelle Nganou Makamdop

    (Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health)

  • Gregory Q. del Prete

    (AIDS and Cancer Virus Program, Leidos Biomedical Research, Inc., Frederick National Laboratory, Frederick, Maryland 21702, USA)

  • Brenna J. Hill

    (Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health)

  • J. Katherina Timmer

    (Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health)

  • Emma Reiss

    (Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health)

  • Ganit Yarden

    (Weizmann Institute of Science, Rehovot 76100, Israel)

  • Samuel Darko

    (Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health)

  • Eduardo Contijoch

    (Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health)

  • John Paul Todd

    (Laboratory of Animal Medicine, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health)

  • Guido Silvestri

    (Emory Vaccine Center, Yerkes National Primate Research Center)

  • Martha Nason

    (Biostatistics Research Branch, National Institute of Allergy and Infectious Diseases, National Institutes of Health)

  • Robert B. Norgren Jr

    (Cell Biology and Anatomy, University of Nebraska Medical Center)

  • Brandon F. Keele

    (AIDS and Cancer Virus Program, Leidos Biomedical Research, Inc., Frederick National Laboratory, Frederick, Maryland 21702, USA)

  • Srinivas Rao

    (Laboratory of Animal Medicine, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health)

  • Jerome A. Langer

    (Rutgers - Robert Wood Johnson Medical School)

  • Jeffrey D. Lifson

    (AIDS and Cancer Virus Program, Leidos Biomedical Research, Inc., Frederick National Laboratory, Frederick, Maryland 21702, USA)

  • Gideon Schreiber

    (Weizmann Institute of Science, Rehovot 76100, Israel)

  • Daniel C. Douek

    (Human Immunology Section, Vaccine Research Center, National Institute of Allergy and Infectious Disease, National Institutes of Health)

Abstract

The timing of type I interferon signalling determines the disease course of SIV infection.

Suggested Citation

  • Netanya G. Sandler & Steven E. Bosinger & Jacob D. Estes & Richard T. R. Zhu & Gregory K. Tharp & Eli Boritz & Doron Levin & Sathi Wijeyesinghe & Krystelle Nganou Makamdop & Gregory Q. del Prete & Bre, 2014. "Type I interferon responses in rhesus macaques prevent SIV infection and slow disease progression," Nature, Nature, vol. 511(7511), pages 601-605, July.
    • Handle: RePEc:nat:nature:v:511:y:2014:i:7511:d:10.1038_nature13554
    DOI: 10.1038/nature13554
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