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Cyclin D1–Cdk4 controls glucose metabolism independently of cell cycle progression

Author

Listed:
  • Yoonjin Lee

    (Dana-Farber Cancer Institute
    Harvard Medical School
    Harvard University)

  • John E. Dominy

    (Dana-Farber Cancer Institute
    Harvard Medical School)

  • Yoon Jong Choi

    (Dana-Farber Cancer Institute
    Harvard Medical School)

  • Michael Jurczak

    (Yale University School of Medicine)

  • Nicola Tolliday

    (Chemical Biology Platform, Broad Institute of Harvard and MIT, 7 Cambridge Center)

  • Joao Paulo Camporez

    (Yale University School of Medicine)

  • Helen Chim

    (Dana-Farber Cancer Institute
    Harvard Medical School)

  • Ji-Hong Lim

    (Dana-Farber Cancer Institute
    Harvard Medical School)

  • Hai-Bin Ruan

    (Yale University School of Medicine)

  • Xiaoyong Yang

    (Yale University School of Medicine)

  • Francisca Vazquez

    (Dana-Farber Cancer Institute
    Harvard Medical School)

  • Piotr Sicinski

    (Dana-Farber Cancer Institute
    Harvard Medical School)

  • Gerald I. Shulman

    (Yale University School of Medicine)

  • Pere Puigserver

    (Dana-Farber Cancer Institute
    Harvard Medical School)

Abstract

Formation of an active cyclin D1–Cdk4 complex suppresses glucose metabolism independently of cell division.

Suggested Citation

  • Yoonjin Lee & John E. Dominy & Yoon Jong Choi & Michael Jurczak & Nicola Tolliday & Joao Paulo Camporez & Helen Chim & Ji-Hong Lim & Hai-Bin Ruan & Xiaoyong Yang & Francisca Vazquez & Piotr Sicinski &, 2014. "Cyclin D1–Cdk4 controls glucose metabolism independently of cell cycle progression," Nature, Nature, vol. 510(7506), pages 547-551, June.
    • Handle: RePEc:nat:nature:v:510:y:2014:i:7506:d:10.1038_nature13267
    DOI: 10.1038/nature13267
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