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Balancing regeneration and cancer

Author

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  • Christian M Beausejour

    (Centre de recherche pédiatrique, Hôpital Sainte-Justine, Université de Montréal, 3175 chemin de la Côte-Sainte-Catherine)

  • Judith Campisi

    (Lawrence Berkeley National Laboratory, and the Buck Institute for Age Research)

Abstract

The proliferation of cells must balance the longevity assured by tissue renewal against the risk of developing cancer. The tumour-suppressor protein p16INK4a seems to act at the pivot of this delicate equilibrium.

Suggested Citation

  • Christian M Beausejour & Judith Campisi, 2006. "Balancing regeneration and cancer," Nature, Nature, vol. 443(7110), pages 404-405, September.
    • Handle: RePEc:nat:nature:v:443:y:2006:i:7110:d:10.1038_nature05221
    DOI: 10.1038/nature05221
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    Cited by:

    1. Albert Stuart Reece & Gary Kenneth Hulse, 2022. "Epigenomic and Other Evidence for Cannabis-Induced Aging Contextualized in a Synthetic Epidemiologic Overview of Cannabinoid-Related Teratogenesis and Cannabinoid-Related Carcinogenesis," IJERPH, MDPI, vol. 19(24), pages 1-57, December.

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