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p53 polymorphism and risk of cervical cancer

Author

Listed:
  • Alan Storey

    (Imperial Cancer Research Fund, Skin Tumour Laboratory)

  • Miranda Thomas

    (International Centre for Genetic Engineering and Biotechnology)

  • Ann Kalita

    (Institute of Parasitology and McGill Cancer Centre, McGill University)

  • Catherine Harwood

    (Imperial Cancer Research Fund, Skin Tumour Laboratory)

  • Daniela Gardiol

    (International Centre for Genetic Engineering and Biotechnology)

  • Fiamma Mantovani

    (International Centre for Genetic Engineering and Biotechnology)

  • Judith Breuer

    (St Bartholomew's and The Royal London Hospital School of Medicine and Dentistry, Queen Mary and Westfield College)

  • Irene M. Leigh

    (International Centre for Genetic Engineering and Biotechnology)

  • Greg Matlashewski

    (Institute of Parasitology and McGill Cancer Centre, McGill University)

  • Lawrence Banks

    (International Centre for Genetic Engineering and Biotechnology)

Abstract

Storey et al. reply — These reports assess the frequency of the p53Arg allele in different populations and conclude that homozygous p53Arg is not a risk factor for cancer associated with human papilloma virus (HPV). The functional differences between the p53 isoforms that we have described1,2 provoked our initial epidemiological study. As we concluded then, it is crucial that investigations should be extended to different populations, and we are encouraged that such studies are underway.

Suggested Citation

  • Alan Storey & Miranda Thomas & Ann Kalita & Catherine Harwood & Daniela Gardiol & Fiamma Mantovani & Judith Breuer & Irene M. Leigh & Greg Matlashewski & Lawrence Banks, 1998. "p53 polymorphism and risk of cervical cancer," Nature, Nature, vol. 396(6711), pages 532-532, December.
    • Handle: RePEc:nat:nature:v:396:y:1998:i:6711:d:10.1038_25043
    DOI: 10.1038/25043
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