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Publisher Correction: Spermine synthase deficiency causes lysosomal dysfunction and oxidative stress in models of Snyder-Robinson syndrome

Author

Listed:
  • Chong Li

    (University of Miami Miller School of Medicine)

  • Jennifer M. Brazill

    (University of Miami Miller School of Medicine)

  • Sha Liu

    (School of Pharmacy, Key Laboratory of Molecular Pharmacology and Drug Evaluation (Yantai University), Ministry of Education, Collaborative Innovation Center of Advanced Drug Delivery System and Biotech Drugs in Universities of Shandong, Yantai University)

  • Christofer Bello

    (University of Miami Miller School of Medicine)

  • Yi Zhu

    (University of Miami Miller School of Medicine)

  • Marie Morimoto

    (NIH Undiagnosed Diseases Program, National Human Genome Research Institute, NIH
    Section of Human Biochemical Genetics, Medical Genetics Branch, National Human Genome Research Institute, NIH)

  • Lauren Cascio

    (JC Self Research Institute, Greenwood Genetic Center)

  • Rini Pauly

    (JC Self Research Institute, Greenwood Genetic Center)

  • Zoraida Diaz-Perez

    (University of Miami Miller School of Medicine)

  • May Christine V. Malicdan

    (NIH Undiagnosed Diseases Program, National Human Genome Research Institute, NIH
    Section of Human Biochemical Genetics, Medical Genetics Branch, National Human Genome Research Institute, NIH
    Office of the Clinical Director, National Human Genome Research Institute, NIH)

  • Hongbo Wang

    (School of Pharmacy, Key Laboratory of Molecular Pharmacology and Drug Evaluation (Yantai University), Ministry of Education, Collaborative Innovation Center of Advanced Drug Delivery System and Biotech Drugs in Universities of Shandong, Yantai University)

  • Luigi Boccuto

    (JC Self Research Institute, Greenwood Genetic Center)

  • Charles E. Schwartz

    (JC Self Research Institute, Greenwood Genetic Center)

  • William A. Gahl

    (NIH Undiagnosed Diseases Program, National Human Genome Research Institute, NIH
    Section of Human Biochemical Genetics, Medical Genetics Branch, National Human Genome Research Institute, NIH
    Office of the Clinical Director, National Human Genome Research Institute, NIH)

  • Cornelius F. Boerkoel

    (NIH Undiagnosed Diseases Program, National Human Genome Research Institute, NIH
    Section of Human Biochemical Genetics, Medical Genetics Branch, National Human Genome Research Institute, NIH
    University of British Columbia)

  • R. Grace Zhai

    (University of Miami Miller School of Medicine
    School of Pharmacy, Key Laboratory of Molecular Pharmacology and Drug Evaluation (Yantai University), Ministry of Education, Collaborative Innovation Center of Advanced Drug Delivery System and Biotech Drugs in Universities of Shandong, Yantai University)

Abstract

The originally published version of this Article contained errors in Figure 1. In panel c, the grey shading denoting evolutionary conservation and the arrowheads indicating amino acids affected in Snyder-Robinson syndrome were displaced relative to the sequence. These errors have now been corrected in both the PDF and HTML versions of the manuscript.

Suggested Citation

  • Chong Li & Jennifer M. Brazill & Sha Liu & Christofer Bello & Yi Zhu & Marie Morimoto & Lauren Cascio & Rini Pauly & Zoraida Diaz-Perez & May Christine V. Malicdan & Hongbo Wang & Luigi Boccuto & Char, 2018. "Publisher Correction: Spermine synthase deficiency causes lysosomal dysfunction and oxidative stress in models of Snyder-Robinson syndrome," Nature Communications, Nature, vol. 9(1), pages 1-1, December.
    • Handle: RePEc:nat:natcom:v:9:y:2018:i:1:d:10.1038_s41467-017-02462-8
    DOI: 10.1038/s41467-017-02462-8
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