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Correction: Corrigendum: Parkinson-causing α-synuclein missense mutations shift native tetramers to monomers as a mechanism for disease initiation

Author

Listed:
  • Ulf Dettmer
  • Andrew J. Newman
  • Frank Soldner
  • Eric S. Luth
  • Nora C. Kim
  • Victoria E. von Saucken
  • John B. Sanderson
  • Rudolf Jaenisch
  • Tim Bartels
  • Dennis Selkoe

Abstract

Nature Communications 6, Article number: 7314 (2015); Published: 16 June 2015; Updated: 30 July 2015. The original version of this Article contained an error in the email address of the corresponding author Dennis Selkoe. The correct email is dselkoe@partners.org. The error has been corrected in theHTML and PDF versions of the article.

Suggested Citation

  • Ulf Dettmer & Andrew J. Newman & Frank Soldner & Eric S. Luth & Nora C. Kim & Victoria E. von Saucken & John B. Sanderson & Rudolf Jaenisch & Tim Bartels & Dennis Selkoe, 2015. "Correction: Corrigendum: Parkinson-causing α-synuclein missense mutations shift native tetramers to monomers as a mechanism for disease initiation," Nature Communications, Nature, vol. 6(1), pages 1-1, November.
    • Handle: RePEc:nat:natcom:v:6:y:2015:i:1:d:10.1038_ncomms9008
    DOI: 10.1038/ncomms9008
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    Cited by:

    1. Cheng Fu & Nan Yang & Jen-Zen Chuang & Nobuyuki Nakajima & Satoshi Iraha & Neeta Roy & Zhenquan Wu & Zhichun Jiang & Wataru Otsu & Roxana A. Radu & Howard Hua Yang & Maxwell Ping Lee & Tilla S. Worgal, 2024. "Mutant mice with rod-specific VPS35 deletion exhibit retinal α-synuclein pathology-associated degeneration," Nature Communications, Nature, vol. 15(1), pages 1-19, December.

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