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Reduced Tyk2 gene expression in β-cells due to natural mutation determines susceptibility to virus-induced diabetes

Author

Listed:
  • Kenichi Izumi

    (Graduate School of Medical Sciences, Kyushu University
    Graduate School of Medical Sciences, Kyushu University
    Diabetes and Endocrinology, School of Medicine, Saga University)

  • Keiichiro Mine

    (Graduate School of Medical Sciences, Kyushu University)

  • Yoshitaka Inoue

    (Graduate School of Medical Sciences, Kyushu University)

  • Miho Teshima

    (Graduate School of Medical Sciences, Kyushu University)

  • Shuichiro Ogawa

    (Graduate School of Medical Sciences, Kyushu University)

  • Yuji Kai

    (Graduate School of Medical Sciences, Kyushu University)

  • Toshinobu Kurafuji

    (Graduate School of Medical Sciences, Kyushu University)

  • Kanako Hirakawa

    (Graduate School of Medical Sciences, Kyushu University)

  • Daiki Miyakawa

    (Graduate School of Medical Sciences, Kyushu University)

  • Haruka Ikeda

    (Graduate School of Medical Sciences, Kyushu University)

  • Akari Inada

    (Graduate School of Medical Sciences, Kyushu University)

  • Manami Hara

    (University of Chicago)

  • Hisakata Yamada

    (Research Center for Prevention of Infectious Diseases, Medical Institute of Bioregulation, Kyushu University)

  • Koichi Akashi

    (Graduate School of Medical Sciences, Kyushu University)

  • Yoshiyuki Niho

    (Graduate School of Medical Sciences, Kyushu University)

  • Keisuke Ina

    (Faculty of Medicine, Oita University)

  • Takashi Kobayashi

    (Faculty of Medicine, Oita University)

  • Yasunobu Yoshikai

    (Research Center for Prevention of Infectious Diseases, Medical Institute of Bioregulation, Kyushu University)

  • Keizo Anzai

    (Diabetes and Endocrinology, School of Medicine, Saga University)

  • Teruo Yamashita

    (Aichi Prefectural Institute of Public Health)

  • Hiroko Minagawa

    (Aichi Prefectural Institute of Public Health)

  • Shuji Fujimoto

    (Graduate School of Medical Sciences, Kyushu University)

  • Hironori Kurisaki

    (Graduate School of Medical Sciences, Kyushu University)

  • Kazuya Shimoda

    (Faculty of Medicine, University of Miyazaki)

  • Hitoshi Katsuta

    (Graduate School of Medical Sciences, Kyushu University)

  • Seiho Nagafuchi

    (Graduate School of Medical Sciences, Kyushu University)

Abstract

Accumulating evidence suggests that viruses play an important role in the development of diabetes. Although the diabetogenic encephalomyocarditis strain D virus induces diabetes in restricted lines of inbred mice, the susceptibility genes to virus-induced diabetes have not been identified. We report here that novel Tyrosine kinase 2 (Tyk2) gene mutations are present in virus-induced diabetes-sensitive SJL and SWR mice. Mice carrying the mutant Tyk2 gene on the virus-resistant C57BL/6 background are highly sensitive to virus-induced diabetes. Tyk2 gene expression is strongly reduced in Tyk2-mutant mice, associated with low Tyk2 promoter activity, and leads to decreased expression of interferon-inducible genes, resulting in significantly compromised antiviral response. Tyk2-mutant pancreatic β-cells are unresponsive even to high dose of Type I interferon. Reversal of virus-induced diabetes could be achieved by β-cell-specific Tyk2 gene expression. Thus, reduced Tyk2 gene expression in pancreatic β-cells due to natural mutation is responsible for susceptibility to virus-induced diabetes.

Suggested Citation

  • Kenichi Izumi & Keiichiro Mine & Yoshitaka Inoue & Miho Teshima & Shuichiro Ogawa & Yuji Kai & Toshinobu Kurafuji & Kanako Hirakawa & Daiki Miyakawa & Haruka Ikeda & Akari Inada & Manami Hara & Hisaka, 2015. "Reduced Tyk2 gene expression in β-cells due to natural mutation determines susceptibility to virus-induced diabetes," Nature Communications, Nature, vol. 6(1), pages 1-10, November.
  • Handle: RePEc:nat:natcom:v:6:y:2015:i:1:d:10.1038_ncomms7748
    DOI: 10.1038/ncomms7748
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    Cited by:

    1. Vikash Chandra & Hazem Ibrahim & Clémentine Halliez & Rashmi B. Prasad & Federica Vecchio & Om Prakash Dwivedi & Jouni Kvist & Diego Balboa & Jonna Saarimäki-Vire & Hossam Montaser & Tom Barsby & Väin, 2022. "The type 1 diabetes gene TYK2 regulates β-cell development and its responses to interferon-α," Nature Communications, Nature, vol. 13(1), pages 1-16, December.
    2. Keiichiro Mine & Seiho Nagafuchi & Satoru Akazawa & Norio Abiru & Hitoe Mori & Hironori Kurisaki & Kazuya Shimoda & Yasunobu Yoshikai & Hirokazu Takahashi & Keizo Anzai, 2024. "TYK2 signaling promotes the development of autoreactive CD8+ cytotoxic T lymphocytes and type 1 diabetes," Nature Communications, Nature, vol. 15(1), pages 1-14, December.

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