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Author Correction: Atrx inactivation drives disease-defining phenotypes in glioma cells of origin through global epigenomic remodeling

Author

Listed:
  • Carla Danussi

    (University of Texas MD Anderson Cancer Center)

  • Promita Bose

    (Memorial Sloan-Kettering Cancer Center)

  • Prasanna T. Parthasarathy

    (Memorial Sloan-Kettering Cancer Center)

  • Pedro C. Silberman

    (Memorial Sloan-Kettering Cancer Center)

  • John S. Arnam

    (University of Texas MD Anderson Cancer Center)

  • Mark Vitucci

    (University of North Carolina School of Medicine)

  • Oliver Y. Tang

    (Memorial Sloan-Kettering Cancer Center)

  • Adriana Heguy

    (New York University School of Medicine)

  • Yuxiang Wang

    (Memorial Sloan-Kettering Cancer Center)

  • Timothy A. Chan

    (Memorial Sloan-Kettering Cancer Center
    Memorial Sloan-Kettering Cancer Center)

  • Gregory J. Riggins

    (Johns Hopkins School of Medicine)

  • Erik P. Sulman

    (University of Texas MD Anderson Cancer Center
    University of Texas MD Anderson Cancer Center)

  • Frederick F. Lang

    (University of Texas MD Anderson Cancer Center)

  • Chad J. Creighton

    (Baylor College of Medicine)

  • Benjamin Deneen

    (Baylor College of Medicine)

  • C. Ryan Miller

    (University of North Carolina School of Medicine
    University of North Carolina School of Medicine)

  • David J. Picketts

    (University of Ottawa
    Ottawa Hospital Research Institute)

  • Kasthuri Kannan

    (New York University School of Medicine)

  • Jason T. Huse

    (University of Texas MD Anderson Cancer Center
    University of Texas MD Anderson Cancer Center)

Abstract

No abstract is available for this item.

Suggested Citation

  • Carla Danussi & Promita Bose & Prasanna T. Parthasarathy & Pedro C. Silberman & John S. Arnam & Mark Vitucci & Oliver Y. Tang & Adriana Heguy & Yuxiang Wang & Timothy A. Chan & Gregory J. Riggins & Er, 2022. "Author Correction: Atrx inactivation drives disease-defining phenotypes in glioma cells of origin through global epigenomic remodeling," Nature Communications, Nature, vol. 13(1), pages 1-1, December.
    • Handle: RePEc:nat:natcom:v:13:y:2022:i:1:d:10.1038_s41467-021-27820-5
    DOI: 10.1038/s41467-021-27820-5
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