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Genome-wide analysis highlights contribution of immune system pathways to the genetic architecture of asthma

Author

Listed:
  • Yi Han

    (University of Southern California
    University of Southern California)

  • Qiong Jia

    (University of Southern California
    University of Southern California)

  • Pedram Shafiei Jahani

    (University of Southern California)

  • Benjamin P. Hurrell

    (University of Southern California)

  • Calvin Pan

    (David Geffen School of Medicine at UCLA)

  • Pin Huang

    (University of Southern California
    University of Southern California)

  • Janet Gukasyan

    (University of Southern California
    University of Southern California)

  • Nicholas C. Woodward

    (University of Southern California
    University of Southern California)

  • Eleazar Eskin

    (University of California, Los Angeles)

  • Frank D. Gilliland

    (University of Southern California)

  • Omid Akbari

    (University of Southern California)

  • Jaana A. Hartiala

    (University of Southern California
    University of Southern California)

  • Hooman Allayee

    (University of Southern California
    University of Southern California)

Abstract

Asthma is a chronic and genetically complex respiratory disease that affects over 300 million people worldwide. Here, we report a genome-wide analysis for asthma using data from the UK Biobank and the Trans-National Asthma Genetic Consortium. We identify 66 previously unknown asthma loci and demonstrate that the susceptibility alleles in these regions are, either individually or as a function of cumulative genetic burden, associated with risk to a greater extent in men than women. Bioinformatics analyses prioritize candidate causal genes at 52 loci, including CD52, and demonstrate that asthma-associated variants are enriched in regions of open chromatin in immune cells. Lastly, we show that a murine anti-CD52 antibody mimics the immune cell-depleting effects of a clinically used human anti-CD52 antibody and reduces allergen-induced airway hyperreactivity in mice. These results further elucidate the genetic architecture of asthma and provide important insight into the immunological and sex-specific relevance of asthma-associated risk variants.

Suggested Citation

  • Yi Han & Qiong Jia & Pedram Shafiei Jahani & Benjamin P. Hurrell & Calvin Pan & Pin Huang & Janet Gukasyan & Nicholas C. Woodward & Eleazar Eskin & Frank D. Gilliland & Omid Akbari & Jaana A. Hartiala, 2020. "Genome-wide analysis highlights contribution of immune system pathways to the genetic architecture of asthma," Nature Communications, Nature, vol. 11(1), pages 1-13, December.
  • Handle: RePEc:nat:natcom:v:11:y:2020:i:1:d:10.1038_s41467-020-15649-3
    DOI: 10.1038/s41467-020-15649-3
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    Cited by:

    1. Pietro Demela & Nicola Pirastu & Blagoje Soskic, 2023. "Cross-disorder genetic analysis of immune diseases reveals distinct gene associations that converge on common pathways," Nature Communications, Nature, vol. 14(1), pages 1-12, December.
    2. Elmo C. Saarentaus & Juha Karjalainen & Joel T. Rämö & Tuomo Kiiskinen & Aki S. Havulinna & Juha Mehtonen & Heidi Hautakangas & Sanni Ruotsalainen & Max Tamlander & Nina Mars & Sanna Toppila-Salmi & M, 2023. "Inflammatory and infectious upper respiratory diseases associate with 41 genomic loci and type 2 inflammation," Nature Communications, Nature, vol. 14(1), pages 1-15, December.

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