IDEAS home Printed from https://ideas.repec.org/a/eur/ejnmjr/87.html
   My bibliography  Save this article

Insulin Resistance; is it an Actual Pathologic Compromise of Cellular Integrity or it is just a Potential Toxic Influence on Cellular Function

Author

Listed:
  • Abdullah Nasrat

    (Zaitona Medical Center, Medina, Saudi Arabia)

Abstract

Aim: Demonstration of a possible co-incident toxic influence on cellular function leading to a potential insulin resistance rather than it is an actual permanent compromise of cellular integrity. Background: The spread of DM is rising all over the world in a dramatic way same as the fire spreading in hey especially in developing countries giving the term "diabetic epidemic" an actual credibility. The late three decades demonstrated grave antibiotic aggression towards the stomach bacterium Helicobacter pylori. H. pylori could be forced to migrate to the colon under the influence of the antibiotic violence with consequent accumulation of profuse toxic amounts of colonic ammonia unopposed or buffered by any acidity leading to a biological toxic stress to the body that could predispose to toxic pancreatitis and stress diabetes among those disadvantaged susceptible population. In the same manner, a toxic influence on cellular function could also develop leading to a sort of insulin resistance. Design: A Prospective multiple-case clinical study. Methods: A potent natural colon clear was done for insulin-dependent diabetic patients developing insulin resistance. Results: Response of most patients for insulin administration markedly recovered. Conclusion: The challenge of insulin resistance might be just a potential toxic influence on cellular function rather it is an actual permanent compromise of cellular integrity.

Suggested Citation

  • Abdullah Nasrat, 2024. "Insulin Resistance; is it an Actual Pathologic Compromise of Cellular Integrity or it is just a Potential Toxic Influence on Cellular Function," European Journal of Natural Sciences and Medicine Articles, Revistia Research and Publishing, vol. 7, 2024.
    • Handle: RePEc:eur:ejnmjr:87
    as

    Download full text from publisher

    To our knowledge, this item is not available for download. To find whether it is available, there are three options:
    1. Check below whether another version of this item is available online.
    2. Check on the provider's web page whether it is in fact available.
    3. Perform a search for a similarly titled item that would be available.

    Corrections

    All material on this site has been provided by the respective publishers and authors. You can help correct errors and omissions. When requesting a correction, please mention this item's handle: RePEc:eur:ejnmjr:87. See general information about how to correct material in RePEc.

    If you have authored this item and are not yet registered with RePEc, we encourage you to do it here. This allows to link your profile to this item. It also allows you to accept potential citations to this item that we are uncertain about.

    We have no bibliographic references for this item. You can help adding them by using this form .

    If you know of missing items citing this one, you can help us creating those links by adding the relevant references in the same way as above, for each refering item. If you are a registered author of this item, you may also want to check the "citations" tab in your RePEc Author Service profile, as there may be some citations waiting for confirmation.

    For technical questions regarding this item, or to correct its authors, title, abstract, bibliographic or download information, contact: Revistia Research and Publishing (email available below). General contact details of provider: https://revistia.com/index.php/ejnm .

    Please note that corrections may take a couple of weeks to filter through the various RePEc services.

    IDEAS is a RePEc service. RePEc uses bibliographic data supplied by the respective publishers.